Posttranslational regulation of RAC/ROP functions in barley disease susceptibility and basal resistance to powdery mildew.

Genetic loss of susceptibility is considered a novel tool in breeding for durable pathogen resistance in crops. A detailed understanding of the mechanism of host susceptibility to avoid pleiotropic effects of recessively inherited resistance is necessary. The barley small monomeric RAC/ROP G-protein RACB is a susceptibility factor in the interaction of barley and the barley powdery mildew fungus Blumeria graminis f.sp. hordei (Bgh). Here, we suggest to study RACB-regulated genes coding for putative plasma membrane localised receptor proteins, which might act upstream of RACB.